DISSERTATION APICAL PERIODONTITIS

Molecular analysis of persistent periradicular lesions and root ends reveals a diverse microbial profile. Pseudoramibacter alactolyticus in primary endodontic infections. Identification and quantification of archaea involved in primary endodontic infections. Of 33 paired samples of resected root ends and apical periodontitis specimens, bacteria were always detected in the former, except for one case. Synergistes clone BA and Bacteroidetes clone X are possibly the most prevalent as-yet-uncultivated phylotypes found in endodontic infections 22 , 28 , 30 , 66 ,

Interpretation of the community behavior can be inferred by global gene expression transcriptomics or by comprehensive inventories of released proteins proteomics and metabolites metabolomics. Detection of Treponema denticola in endodontic infections by 16S rRNA gene-directed polymerase chain reaction. This panel of selected pathogens can then be used against large numbers of clinical samples with reduced cost and time to identify associations with specific disease forms, signs, and symptoms. Bacteriological study of root canals associated with periapical abscesses. The recognition that the microbiota associated with the same endodontic disease significantly differs in species richness and abundance between individuals living in different geographic locations 18 , raises the inevitable question as to whether the same treatment protocols, especially systemic antibiotic therapy, can be used in one-size-fits-all terms. Effects of instrumentation, irrigation and dressing with calcium hydroxide on infection in pulpless teeth with periapical bone lesions. Molecular epidemiology and association of putative pathogens in root canal infection.

After making a selection, click one of the export format buttons. Bacteria in the apical root canal of teeth with primary apical periodontitis. This review paper highlights the distinctive features of the endodontic microbiota associated with diverse clinical conditions.

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Based on cross-sectional studies, several species have emerged as candidate or putative endodontic pathogens, and the labels candidate or putative should be periodonittis while a definite role in disease causation is not determined. Reduced diversity of faecal microbiota in Crohn’s disease revealed by a metagenomic approach.

dissertation apical periodontitis

apjcal Acute apical periodontitis and abscesses Acute apical periodontitis and acute apical abscesses are typical acute forms of apical periodontitis. On the other hand, previous culture and microscopic studies have failed to detect microorganisms in some cases of root canal-treated teeth with persistent disease 99, PCR-based identification of bacteria associated with endodontic infections.

  METODE THINKING ALOUD PAIR PROBLEM SOLVING (TAPPS) ADALAH

Apical actinomycosis, which is caused by Actinomyces species or P.

Distinctive features of the microbiota associated with different forms of apical periodontitis

Spirochetes comprise another bacterial group that has been shown to have as-yet-uncultivated representatives in primary infections. Of 33 paired samples of resected root ends and apical periodontitis specimens, bacteria were always detected in the former, except for one case.

Influence of residual bacteria on periapical tissue healing after chemomechanical treatment and root filling of experimentally infected monkey teeth.

All these findings indicate that the microbiota of root canal-treated teeth with apical periodontitis is more complex than previously anticipated by culture studies. These factors possibly include: This species has been considered as transient in the oral cavity and its source might be food Fusobacterium nucleatum in endodontic flare-ups. In other words, while associations of any specific species with any form of apical periodontitis is seldom, if ever, observed, the bacterial community profiles seem to follow some patterns related to the different presentations of apical periodontitis Although dsisertation transition from acute apical periodontitis to abscess can make it difficult to clinically distinguish these two conditions, in later stages of the disease process the diagnosis of acute abscesses usually does not represent a difficult task, mostly because of swelling.

Distinctive features of the microbiota associated with different forms of apical periodontitis

Search and download doctoral PHD dissertations from Sweden. Identification and potential mechanisms of resistance to antimicrobial measures. Apical periodontitis lesions usually represent an effective immunological barrier against spread of the infection from the root canal periodontitsi the alveolar bone and other body sites.

Acute apical periodontitis and acute apical abscesses are typical acute forms of apical periodontitis. Journal List J Oral Microbiol v. The latter can be explained by the different physico-chemical conditions and type of nutrient availability in the different regions of the canal.

  CURRICULUM VITAE VZOR ANGLI TINA

Molecular identification of microorganisms from endodontic infections.

The Association of Statin Intake and Healing of Apical Periodontitis After Root Canal Treatment

Identification of Enterococcus faecalis in root-filled teeth with or without periradicular lesions by culture-dependent and -independent approaches. Intraradicular bacteria and fungi in root-filled, asymptomatic human teeth with therapy-resistant periapical lesions: As for flare-ups, the evidence of specificity is even weaker, although there are some reports of involvement apjcal gram-negative anaerobes, such as black-pigmented rods and F.

Support Center Support Center. Ricucci D, Siqueira JF.

dissertation apical periodontitis

Persistent infections are caused by microorganisms that were members of a primary or secondary infection and that persisted in the canal after antimicrobial treatment and managed to adapt to the harsh ecological conditions in instrumented and filled root canals.

In this regard, further studies exploring a holistic view of endodontic infections are made necessary, where the species colonizing the untreated or treated canal should be seen as part of an ecosystem, in which they live in communities whose outcome of physiology and function will determine the outcome of the tissue response, i. This indicates that, even if total bacterial elimination is not the case, at least a substantial reduction in species richness and abundance is attained.

dissertation apical periodontitis

Herpesviruses in abscesses and cellulitis of endodontic origin. Patterns of dissertatjon colonization in primary root canal infections. The last years have witnessed a trend toward a more holistic concept of the etiology of human endogenous infections that considers the bacterial community as the unit of pathogenicity Molecular epidemiology and association of putative pathogens in root canal infection.